X-linked dystonia-parkinsonismResearch
Recent research highlights a non-canonical function of BRD4 in correcting the molecular phenotype of X-linked Dystonia-Parkinsonism. This study contributes to the understanding of the disease's underlying mechanisms.
correction of the molecular phenotype of x linked dystonia parkinsonism reveals a non canonical function of brd4
Original title: “Correction of the molecular phenotype of X-linked Dystonia-Parkinsonism reveals a non-canonical function of BRD4.”