Research demonstrates that BIN1 overexpression can rescue cardiac defects in a mouse model of caveolinopathy, but does not improve skeletal muscle defects. This study provides insights into potential therapeutic targets for cardiac issues associated with caveolinopathy.
bin1 overexpression rescues cardiac but not skeletal muscle defects in a mouse model of caveolinopathy
Original title: “BIN1 overexpression rescues cardiac but not skeletal muscle defects in a mouse model of caveolinopathy.”